Semax 10mg Peptide | Third-Party Tested | RemedyX Labs
Order Semax 10mg research peptide online from RemedyX Labs. Verified batch COAs confirm purity and identity. Secure checkout, competitive pricing, and fast shipping.
Semax is a synthetic peptide derived from a fragment of adrenocorticotropic hormone (ACTH 4-10), studied in controlled laboratory models of neuropeptide signaling, BDNF/TrkB pathway activity, and neurological research. Every batch is independently tested by a third-party laboratory to confirm identity and purity, with batch-specific Certificates of Analysis (COA) available for full transparency. For laboratory research use only. Not for human or animal use. Not intended for diagnostic, therapeutic, clinical, or commercial use.
- Price: 39.00 USD
- In stock
- Purity: 99%+
- CAS Number: 80714-61-0
- Molecular Formula: C37H51N9O10S
- Molecular Weight: 813.9
- Specification: 10mg/vial
- Contents: Lyophilized Powder
- Appearance: White to Off-White
- Intended Use: Research Only
- SKU: RXL-SKU-0038
Cited Research
Semax, an Analog of ACTH(4-10), Regulates BDNF
This study investigates how the ACTH analog Semax influences the regulation of brain-derived neurotrophic factor.
PubMed
The Peptide Semax Affects the Expression of Genes Related to the Immune and Cardiovascular Systems
Researchers examined the influence of the peptide Semax on gene expression profiles associated with the immune and cardiovascular systems.
PubMed
[The efficacy of semax in the treatment of patients at different stages of ischemic stroke]
This study assessed the efficacy of the peptide Semax in treating patients at various stages of ischemic stroke recovery.
PubMed, 2018
[Effectiveness of semax in acute period of hemispheric ischemic stroke (a clinical and electrophysiological study)]
This non-randomized clinical study used EEG and evoked-potential monitoring to evaluate Semax's effect on neurological recovery during the acute period of hemispheric ischemic stroke.
PubMed, 2001
Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents
This research explored the capacity of the ACTH(4-10) analogue Semax to activate dopaminergic and serotoninergic brain systems in rodent models.
PubMed, 2006
Effects of Semax on the Default Mode Network of the Brain
Scientists utilized fMRI to investigate the influence of Semax administration on the brain's default mode network.
PubMed, 2018
Semax, an analog of ACTH(4-7), regulates expression of immune response genes during ischemic brain injury in rats
This study investigated how the ACTH(4-7) analogue Semax regulates the expression of immune response genes following ischemic brain injury in rats.
PubMed, 2017
Heptapeptide semax attenuates the effects of chronic unpredictable stress in rats
Researchers examined the potential of the heptapeptide Semax to attenuate physiological and behavioral effects induced by chronic unpredictable stress in rats.
PubMed, 2014
Antidepressant-like and antistress effects of the ACTH(4-10) synthetic analogs Semax and Melanotan II on male rats in a model of chronic unpredictable stress
This study compared the antidepressant-like and antistress effects of synthetic ACTH(4-10) analogs Semax and Melanotan II in a rat model of chronic stress.
PubMed, 2024
Semax peptide targets the μ opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female mice
This investigation found that Semax promotes functional recovery after spinal cord injury in female mice by targeting the Oprm1 opioid receptor gene.
PubMed, 2025
The peptide Semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia
Reports changes in genes related to vascular and immune pathways after Semax.
PubMed, 2014
Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Amyloid Aggregation
Examines Semax interactions with copper and amyloid-related processes in vitro.
PMC, 2022
The effect of Semax and its C-end peptide PGP on nervous tissue in brain ischemia
Reports cellular and vascular changes in normal and ischemic brain tissue.
PubMed, 2011
Novel Insights into the Protective Properties of ACTH(4-7)PGP Peptides
Reviews mechanisms and clinical history related to Semax-family peptides.
PMC, 2020
The Potential of the Peptide Drug Semax and Its Derivative for Alzheimer's Disease
Reports improved behavioral measures and reduced amyloid inclusions in a mouse model.
PubMed, 2025
Semax and PGP influence neuroglial, endothelial and progenitor-cell proliferation after ischemia
Reports histologic changes consistent with trophic and vascular effects in an ischemia model.
PubMed, 2011